Re: Experimental basis for the Non-Beneficial Gap Problem
- From: Rupert Morrish <rupert@xxxxxxxxxxx>
- Date: Wed, 09 Jul 2008 11:53:18 +1200
Seanpit wrote:
On Jul 7, 5:28 pm, Rupert Morrish <rup...@xxxxxxxxxxx> wrote:Seanpit wrote:
[snip tornado in a junkyard argument]
Congratulations. You've proved that proteins are not formed by randomly
appending amino acids.
Perhaps next time you should attempt to prove that the sky is not pink?
You obviously don't understand the significance of the argument. It
is a sort of toronado-in-a-junkyard argument - with a twist ; )
What is interesting about this particular version, is that the
relative rarity of potentially beneficial proteins in sequence space
is not only estimatable to a useful degree, this rarity gets
exponentially more and more rare at higher and higher levels of
functional complexity. What that means is that the non-beneficial
gaps between potentially beneficial proteins increases in a linear
manner with each increase in the minimum size and/or specificity
requirement.
I hate to get all backspace on you, but who sets the requirements? Who requires that bacteria be mobile?
And, what that means is that the average number of
mutations needed to cross the gap increases exponentially.
But the minimum number, which is what defines the most likely path, does not.
You may not comprehend the significance of this argument given that
you didn't understand the coded nature of genetic information, but
this argument does in fact fundamentally undermine the proposed
mechanism behind the ToE - i.e., random mutation and natural
selection.
I understand the argument. You are making a statistical argument based on a flawed model of evolution to show that that which has been directly observed can not happen.
If anyone considered that the likelihood of 1000aa protein systems arising without precursors was an argument in favor of evolution, you may have a point. But no-one does, so you are simply attacking a straw man.
Sean Pitman
www.DetectingDesign.com
.
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