Re: A Cure For Cancer? Eating A Plant-Based Diet
- From: crisology <crisology@xxxxxxx>
- Date: Sat, 3 Oct 2009 12:53:45 -0700 (PDT)
On Sep 29, 10:02 am, "D. C. Sessions" <d...@xxxxxxxxxxxxxxxx> wrote:
In message <WDnwm.30177$j7.493...@xxxxxxxxxxxxxx>, O.pearl wrote:
A Cure For Cancer? Eating A Plant-Based Diet
Therefore obligate herbivores (e.g. elk) don't get cancer.
Oh, wait ...
Where is a source for elk cancer rates compared to human cancer rates?
From the cursory research I've done on the topic, I find littleinformation on wild population cancer rates but the available evidence
I've read seems to indicate that cancer (among other acquired
diseases) is rare among wild animals (especially those not exposed to
human pollutions ie chromium, fluoride, etc.).
"This would appear to be the first report of an oligodendroglioma in a
deer."
Journal of Comparative Pathology Volume 138, Issue 1, January 2008,
Pages 59-62.
"This is the first report of intestinal adenocarcinoma in lions."
European Journal of Wildlife Research, Volume 54, Number 2, May 2008 ,
pp. 385-389(5)
etc..
The lion of course eats meat (and could have relatively less cancer)
but there is little doubt (or is there?) lions are better adapted to
digest meat.
While raw and cooked meat consumption provide different ways to
acquire cancer for humans (Meats can cause cancer by several routes,
the researchers noted. "For example, they are both sources of
saturated fat and iron, which have independently been associated with
carcinogenesis," U.S. National Cancer Institute Library of Science
journal PLoS Medicine. 2007.), meat consumption isn't the only way to
acquire cancer. Genetic predisposition may play a smaller role but a
role, nonetheless.
Pharm Res. 2008 Jul 15
"5-10% of all cancer cases can be attributed to genetic defects..The
evidence indicates that of all cancer-related deaths, almost 25-30%
are due to tobacco, as many as 30-35% are linked to diet, about 15-20%
are due to infections..cancer prevention requires smoking cessation,
increased ingestion of fruits and vegetables, moderate use of alcohol,
caloric restriction, exercise, avoidance of direct exposure to
sunlight, minimal meat consumption, etc.
It seems plant based diets help prevent cancer and there is some
evidence that it also cures cancer to varying degrees.
"Environment determines the risk of both prostate and breast cancer,
and this risk can vary > 10-fold... There is also species specificity,
because there is no risk for prostate cancer in any other aging mammal
except the dog. A study of evolution indicates that the prostate and
breast appeared at the same time 65 million years ago with the
development of mammals. All male mammals have a prostate; however, the
seminal vesicles are variable and are determined by the diet so that
species primarily eating meat do not have seminal vesicles. The
exception is the human, who has seminal vesicles and consumes meat,
although this is a recent dietary change. Human lineage departed from
other higher primates 8 million years ago. The closest existing
primate to humans is the bonobo (pigmy chimpanzee), which does not eat
meat but exists primarily on a high fruit and fresh vegetable diet.
Homo sapiens evolved only about 150,000 years ago, and only in the
last 10% of that time (10 to 15 thousand years ago) did humans and
dogs dramatically alter their diets. This is the time when humans
domesticated the dog, bred animals, grew crops, and cooked, processed,
and stored meats and vegetables. All current epidemiologic evidence
and suggestions for preventing prostate and breast cancer in humans
indicates that we should return to the original diets under which our
ancestors evolved. The recent development of the Western-type diet is
asociated with breast and prostate cancer througout the world. It is
believed that the exposure to and metabolism of estrogens, and the
dietary intake of phytoestrogens, combined with fat intake, obesity,
and burned food processing may all be related to hormonal
carcinogenesis and oxidative DNA damage. An explanatory model is
proposed. Urology 57 (Suppl 4A): 31-38. 2001. Elsevier Science Inc.
There has not been sufficient time for biological selection to evolve
a proper defense for human DNA against the insults perpetuated by the
modern Western diet. This may result in nutritional diseases such as
prostate and breast cancer. Both prostate cancer and breast cancer
primarily occur later in life, often after the optimum age for
reproduction, thus severely limiting the evolutionary selection
process against cancer. This medical dilemma appears to be enhanced by
the speed of technology that enhances the imbalance between the rapid
rate of changes in our food processing and dietary patterns, which far
exceeds the slower rate of evolution of adequate biological defenses.
The interplay of many dietary components, such as phytoestrogens, can
induce hormonal changes that may contribute to the carcinogenic
insults of our lifestyle on prostate and breast tissue. Insights into
the development of the concept presented here have been derived fro
mattempts to forumlate plausible biological explanations of the
following features and enigmantic phenomena observed in prostate and
breast oncology:
1. Species specificity of prostate cancer: A significant incidence of
spontaenous prostate cnacer is observed only in humans and the dog. Of
the thousands of other species of mammals with prostates, none that
age in zoos or captivity have a significant incidence of prostate
cancer that results in clinical diagnoses, metastasis, and death.
Except for the dog, prostate cancer is also absent or extremely rare
in all aging pets such as cats, hamsters, guinea pigs, and horses.
2. Tissue specificity of cancer: Prostate cancer is most common in
humans; however, there ar eother adjacent reproductive organs that
never present with cancers such as the vas deferens, epiddymis, and
bulbourethral gland, or even the seminal vesicles, where it is
extremely rare. In a given individual, all of these glands share
similar genetics, age, and environments but yet have vast differences
in their cancer rates. Neither genetics nor environment is sufficient
to cause bulbourethral, epididymal, or vas deferns cancer in a man. At
present, no molecular explanation is proposed to explain this
selectivity.
3. Geographical variations: Compared with the United States, the
incidence and age-adjusted rates of both breast and prostate cancer
can be >10-fold lower in Asian countries. When Asians migrate to the
United States, their prostate and brast cancer rates tend to incrase,
with time, toward levels seen in the native US population. Although
this seems to strongly implicate environmental factors, a precise
explanation for this phenomenon is not currently available.
4. Similarities in epidemiologic factors: There appear to be very
similar lifestyle risk factors accompanying both prostate and breast
cancer, including a lower risk associated with high intake of fruits,
vegetables, fiber, and soy products and, alternativvely, a higher risk
associated with increased intake of red meat, animal fats, dairy
products, and steroid exposure, as well as body mass and birth weight.
This similarity does not exist with other cancers, such as stomach
cancer.
5. Early events are common: In contrast to clinical cancers, small
incidental foci of prostate cnacer, as well as early pathologic
changes and benign prostateic hyperplasia, appear to have relatively
high incidences with aging in both the United States and Asia. This
suggests that promotion and not initiation may be the critical
difference between the clinical rates of prostate cancer in different
geographical areas.
6. Similarities between prostate and breast: Both glands have many
similarities with regard to physiology, endocrinology, benign tumors,
and the age-adjusted incidence and mortality rates of cancer that
appear to be correlated in various countries.
.... (p. 35) It is recognized that all of the thousands of different
species of male mammals have a prostate gland; thus, this is a common
denominator. However, mammals that eat meat, such as dogs, cats, and
sea lions, do not have seminal vesicle whereas those that eat
primarily plants, such as the great apes, horses, bulls, and rodents,
do have a seminal vesicle. It is apparent that during evolution, diet
is associated in some way with the selection for the presence or
absence of the seminal vesicle. Can diet also be involved in why
humans do not get seminal vesicle cancer but do get prostate cancer?
What is confusing about the above analogy is that the human appears to
be an exception in that we eat large quantities of meat but we still
have a seminal vesicle. This may be the problem. This apparent
exception may be the result of a very recent change and diversion in
our diet and food processing from the plant and fruit diet used over
th emain part of human evolution."
http://physics.cancer.gov/ps2/pdf/evolution/26%20Similarities.pdf
Other common diseases related to meat consumption also seem to be less
prevalent in the closest relatives within their ecological niche,
eating natural diets.
LDL increased experimentally w/atherogenic diets in chimps. -
Vastesaeger et al. 1975.
Raising blood levels of chol produces atherosclerosis in chimps. -
Steinberg.
Oxidized LDL is initiating factor.
Chimps rarely get hyperlipidemia in wild but easily get it from animal
husbandry food. (Doucet et al. 1994).
Chimps are more sensitive to hypercholesterolemia than most primates.
Steinetz et al 1996.
"lipoprotein classes..blood chemistry of chimps & gorillas is very
similar to humans. -Steinetz
“xenotropic constituents from soy, milk, fish, grains in chimp chow..
wild chimps are rarely obese.–Wrangham, 1975. Young captive chimps
have died from myocardial infarction. Blaton, Exp Mol Pathol, 20;
132).
Chimps are ~4.03 xs more sensitive to increased dietary chol than
humans. –Simon Steven Institute Brugge, Belgium (Blaten et al, 1970,
72, 74, Howard et al, 72, Peeters, et al 75, 70 etc)
“Chimps live largely on fruits and leaves and their intake of
nutrients such as Vitamin C is many times that of most of us. They eat
a little meat and hunt monkeys on occasion but they are not well
adapted to meat eating; a small amount of saturated fat sends their
cholesterol levels very high” (Mestel 2002; A20).
“There is no evidence that meat is needed for normal pregnancy or
nursing” (Goodall, 1986, Stanford 1998)
"Deficiency diseases have not been identified for any wild primate
population (Kerr 1972, Wolf 1972). Known cases of nutritional diseases
among primates are laboratory-induced features of captive animals or
culturally induced human diseases. BioScience, Volume 28, Pages
761-766, 1978"
With all the confounding variables and impracticality of using human
subjects for controlled trials, 100% 'proof' is not available for most
disease causes, especially cancer. But careful diet experimentation is
a method of obtaining observation of differences in digestion and
disease risks with long-term natural human diets excluding meat/dairy/
grains/candy/soda, etc.
Since increasing raw fruit and slashing protein intake, including meat
and dairy (filled with natural and synthetic hormones/carcinogens),
I've sloughed off fibroepithelial polyps and they have stopped
propagating. Perhaps the reduced protein and hormones did not allow
for uncontrollable skin proliferation and this could also be the case
with potential malignant tumors. Of course this is not by itself
absolute proof that meat consumption is a cause of cancer but I apply
what I learn from experience and research (available evidence) while
monitoring overall health.
Chris
.
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