Re: Dr Cree...

I do know a few things about the P53 gene, Most neurological disorders
have an inactivation of the p53, while cancer has an over expression
of the p53 gene.

While the "oncogene" idea may not be current, there is, of course, a
genetic aspect to cancer, particularly the p53 gene. Another topic
I'm confident Cee knows nothing about.


Now how mercury *can* cause problems on the p53 gene, ranging from
cancer to neurological disorders...It depends on how mercury has
damaged the gene, if there is partial damage you will land up with an
over expression(which is cancer) Complete damage of that gene will
cause an inactivation(Neurological problems)

How mercury causes cancer


Since at least 1997 we have known that mercury chloride cause gene
overexpression of the c-fos gene which also occurs in various types of
cancers. Another cancer gene that mercury has an impact on is the jun
gene and we have known that from at least 2000. Chromosomal
abnormalities are associated with cancers. In fact that is one of
theories of how cancer starts. We have known for decades that
methymercury and other mercury compounds can cause that. In fact we
have population studies that show blood cells have DNA damage due to
people exposed to mercury compounds including methylmercury. We also
have population studies which show areas that had methylmercury
poisoning had higher cancer rates. All the poisoning did was show
that methylmercury is one of the factors in the normal cancer rate of
cancer.


http://community.oceana.org/?op=displaystory;sid=2007/8/21/15827/3946


On Dec 13, 11:38 pm, wri...@xxxxxxxxxxxxxxxxx (David Wright) wrote:
In article <1194917257.068066.323...@xxxxxxxxxxxxxxxxxxxxxxxxxxxx>,





<drcee...@xxxxxxxxxxxxx> wrote:
On Nov 12, 5:55 pm, Kevysmom <bluebun...@xxxxxxxxx> wrote:
I was being nosy and peeked at your profile. :o)

I see that you have done research for cancer, what causes cancer?

I have a dear friend who is a Professor of Molecular Science, He is
the best and has helped me understand
how our genes play a role in diseases. What I mean by this, If we
inject a toxin into a human body it can target a specific gene and
turn it completely off, or it could just damage it enough where you
would get an over-expression on that gene. This is what happens with
macrocephaly and microcephaly. Could this be what happens regarding
cancer, could there be an over-expression of a protein on a gene that
was slightly damaged from a toxin? What makes a tumor grow?

TIA

Donna

Wow, what a question. What causes cancer?

Within allopathic medicine, they have absolutely no idea. There does
not exist any model of cancer formation to discuss. We do know that
carcinogens and radiation causes cancer. We know that we can induce
tumors by many means. Even palpating a female rat incorrectly can
cause tumors. But, do we have a model for cancer formation...and even
as important, a model for the healing of cancer...no, no, and no.
Modern medicine has no idea why chemo works, even as poorly as it
does.

Of course they do. Cancer cells have high metabolic activity but are
relatively fragile in some ways, thus making them more vulnerable to
the chemo drugs.

Consider the orthopathic model for cancer. This model is what
convinced me to begin a study of this theory of disease.
The body strives for health. If the body cannot provide health for
the host, it provides a longer life. The orthopathic model of disease
shows seven stages of body degeneration: Enervation, toxemia,
irritation, inflammation, ulceration, and induration of the tissues.

If this was enough to convince you, well that says it all.

While the "oncogene" idea may not be current, there is, of course, a
genetic aspect to cancer, particularly the p53 gene. Another topic
I'm confident Cee knows nothing about.

-- David Wright :: alphabeta at prodigy.net
These are my opinions only, but they're almost always correct.
"Saddam Hussein was a bad man, connected with some incredibly dangerous
people: Cheney, Rumsfeld, George Galloway." -- Marcus Brigstocke- Hide quoted text -

- Show quoted text -

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